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K74.69
ICD-10-CM
Cirrhosis Secondary to NASH

Learn about the diagnosis of Cirrhosis Secondary to NASH, including clinical documentation and medical coding for Liver Cirrhosis due to NASH. This guide covers NASH-related Cirrhosis and Non-alcoholic Steatohepatitis Cirrhosis, providing information relevant for healthcare professionals and coding specialists seeking accurate terminology for optimal clinical care and reimbursement.

Also known as

Liver Cirrhosis due to NASH
NASH-related Cirrhosis
Non-alcoholic Steatohepatitis Cirrhosis

Diagnosis Snapshot

Key Facts
  • Definition : Advanced scarring of the liver due to fat buildup and inflammation, not related to alcohol.
  • Clinical Signs : Fatigue, jaundice, swelling, easy bruising, ascites, hepatic encephalopathy.
  • Common Settings : Hepatology clinics, gastroenterology departments, primary care offices.

Related ICD-10 Code Ranges

Complete code families applicable to AAPC K74.69 Coding
K70-K77

Diseases of liver

Covers various liver diseases including cirrhosis.

K74

Fibrosis and cirrhosis of liver

Specifically addresses liver fibrosis and cirrhosis.

K75-K76

Other inflammatory liver diseases

Includes other inflammatory conditions affecting the liver.

Code-Specific Guidance

Decision Tree for

Follow this step-by-step guide to choose the correct ICD-10 code.

Is the cirrhosis definitively due to NASH?

  • Yes

    Is there compensated or decompensated cirrhosis?

  • No

    Is NASH present but not confirmed as the cause of cirrhosis?

Code Comparison

Related Codes Comparison

When to use each related code

Description
Scarring of the liver due to NASH.
Fatty liver disease, not due to alcohol.
Liver inflammation and damage due to NASH.

Documentation Best Practices

Documentation Checklist
  • Document NASH diagnosis supporting cirrhosis.
  • Evidence of fibrosis/imaging showing cirrhosis.
  • Exclude other cirrhosis causes (alcohol, viral).
  • Liver function tests and biopsy findings.
  • Specify decompensation stage (if applicable).

Coding and Audit Risks

Common Risks
  • Unspecified Etiology

    Coding NASH cirrhosis without specifying the underlying non-alcoholic fatty liver disease can lead to undercoding and lost revenue.

  • Clinical Validation

    Insufficient documentation to support the diagnosis of NASH cirrhosis may cause denials and compliance issues. CDI can help ensure accurate coding.

  • Comorbidity Coding

    Failing to capture common comorbidities like hepatocellular carcinoma or hepatic encephalopathy with NASH cirrhosis can affect reimbursement and quality metrics.

Mitigation Tips

Best Practices
  • Weight loss via diet and exercise improves NASH cirrhosis.
  • Diabetes, hypertension management crucial in NASH cirrhosis care.
  • Alcohol abstinence is essential for all cirrhosis patients.
  • Regular liver screenings, imaging vital for NASH cirrhosis.
  • Medication can address liver inflammation, fibrosis in NASH.

Clinical Decision Support

Checklist
  • 1. Verify imaging (ultrasound, CT/MRI) shows cirrhosis.
  • 2. Confirm NASH diagnosis via biopsy or non-invasive methods.
  • 3. Exclude other causes of cirrhosis (alcohol, viral hepatitis).
  • 4. Document NASH-cirrhosis link and severity for accurate coding (K74.6).
  • 5. Assess/manage complications (ascites, encephalopathy) per guidelines.

Reimbursement and Quality Metrics

Impact Summary
  • Medical billing: Accurate ICD-10-CM K74.6 coding for NASH cirrhosis maximizes reimbursement.
  • Coding accuracy: Correct NASH cirrhosis diagnosis coding impacts DRG assignment and hospital case mix index.
  • Hospital reporting: Precise NASH cirrhosis coding improves quality metrics and public health data accuracy.
  • Reimbursement impact: Complete documentation of NASH cirrhosis severity supports appropriate resource utilization and payment.

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Frequently Asked Questions

Common Questions and Answers

Q: What are the most effective strategies for diagnosing NASH cirrhosis in patients with suspected non-alcoholic fatty liver disease?

A: Diagnosing NASH cirrhosis requires a multi-faceted approach. While liver biopsy remains the gold standard for confirming NASH, its invasive nature necessitates exploring non-invasive alternatives. These include advanced imaging techniques like transient elastography (FibroScan) and magnetic resonance elastography (MRE) to assess liver stiffness and fibrosis, often coupled with serum biomarkers such as the Enhanced Liver Fibrosis (ELF) test or the NAFLD Fibrosis Score. It's crucial to consider a patient's clinical presentation, including metabolic syndrome components, and rule out other causes of liver disease. Explore how combining these non-invasive methods can improve diagnostic accuracy and reduce the need for biopsy in patients with suspected NAFLD-related cirrhosis.

Q: How can I differentiate NASH cirrhosis from other etiologies of cirrhosis in a patient presenting with advanced liver disease?

A: Differentiating NASH cirrhosis from other forms, like alcoholic cirrhosis or viral hepatitis-related cirrhosis, hinges on a thorough patient history, including alcohol consumption, medication use, and risk factors for viral hepatitis. Serological testing for viral hepatitis (B, C, and others) is essential. Assessing for features of metabolic syndrome, like obesity, diabetes, and dyslipidemia, can strengthen suspicion for NASH cirrhosis. Imaging characteristics on ultrasound, CT, or MRI may also provide clues. Given the potential overlap in clinical presentation, consider implementing a comprehensive diagnostic algorithm incorporating both laboratory and imaging findings to accurately pinpoint the etiology and tailor management accordingly. Learn more about the emerging role of novel biomarkers in improving diagnostic specificity.

Quick Tips

Practical Coding Tips
  • Code K74.6 NASH, then K70.3 for cirrhosis
  • Query physician for etiology clarity
  • Document NASH progression to cirrhosis
  • Review liver biopsy findings for NASH
  • Consider secondary codes for complications

Documentation Templates

Patient presents with complaints consistent with cirrhosis secondary to non-alcoholic steatohepatitis (NASH).  Symptoms include fatigue, abdominal discomfort, and occasional pruritus.  Physical examination reveals hepatomegaly and mild splenomegaly.  Laboratory findings demonstrate elevated liver enzymes (AST, ALT),  low albumin, prolonged prothrombin time (PT), and increased bilirubin.  Imaging studies, including abdominal ultrasound and transient elastography, suggest the presence of cirrhosis with evidence of hepatic steatosis and fibrosis.  The patient denies significant alcohol consumption, and other causes of chronic liver disease, such as viral hepatitis and autoimmune hepatitis, have been ruled out through serological testing.  Diagnosis of NASH cirrhosis is established based on clinical presentation, laboratory data, imaging findings, and exclusion of other etiologies.  Treatment plan includes lifestyle modifications, focusing on weight loss, dietary changes, and increased physical activity.  Pharmacological interventions for NASH-related fibrosis are being considered and will be discussed with the patient.  Regular monitoring of liver function tests, imaging surveillance for hepatocellular carcinoma, and management of potential complications of cirrhosis, such as ascites and hepatic encephalopathy, will be implemented.  Patient education regarding the progression of liver disease, importance of adherence to treatment, and prognosis has been provided.  ICD-10 code K74.6, non-alcoholic steatohepatitis with fibrosis, is recorded, reflecting the underlying etiology of the cirrhosis.