Understanding Hypervitaminosis D diagnosis, symptoms, and treatment is crucial for healthcare professionals. This resource provides information on clinical documentation, medical coding (ICD-10-CM E67.3), laboratory findings, and best practices for managing Vitamin D toxicity. Explore effective strategies for diagnosis and treatment of elevated vitamin D levels, along with relevant differential diagnoses and complications. Learn about appropriate terminology and coding guidelines for accurate healthcare record-keeping related to Hypervitaminosis D.
Also known as
Hypervitaminosis D
Excessive vitamin D intake or absorption.
Other hypervitaminosis
Excessive levels of vitamins other than A or D.
Disorders of calcium metabolism
Conditions related to abnormal calcium levels, sometimes linked to vitamin D.
Abnormal findings of blood chemistry
May include elevated calcium related to hypervitaminosis D.
Follow this step-by-step guide to choose the correct ICD-10 code.
Is hypervitaminosis D due to excessive intake of vitamin D?
Yes
Any documented manifestation?
No
Is it due to other causes (e.g., disease)?
When to use each related code
| Description |
|---|
| High vitamin D levels |
| Vitamin D toxicity |
| Hypercalcemia |
Using unspecified hypervitaminosis D codes (E64.9) when more specific documentation supports E64.0-E64.8, impacting reimbursement and data accuracy.
Insufficient documentation linking hypervitaminosis D to external causes (e.g., excessive intake) leading to coding errors and compliance issues.
Overlooking related conditions like hypercalcemia or renal impairment as secondary diagnoses, impacting severity capture and quality reporting.
Q: What are the most reliable diagnostic tests to confirm Hypervitaminosis D in adults, and what cutoff values should clinicians use to interpret the results?
A: Diagnosing hypervitaminosis D relies primarily on measuring serum 25-hydroxyvitamin D [25(OH)D] levels. While no universally agreed-upon cutoff exists for hypervitaminosis D, concentrations consistently above 150 ng/mL (375 nmol/L) are generally considered indicative of excessive vitamin D levels. Some experts suggest a lower threshold of 125 ng/mL (312 nmol/L) for potential toxicity. It is crucial to correlate elevated 25(OH)D levels with clinical manifestations and consider other contributing factors like granulomatous diseases or certain medications before confirming a diagnosis. Furthermore, assessing serum calcium, phosphorus, and parathyroid hormone (PTH) levels can help evaluate the impact of excessive vitamin D on calcium homeostasis and identify potential complications. Explore how serial 25(OH)D measurements can assist in monitoring treatment response and preventing recurrence.
Q: How does hypervitaminosis D differ clinically from vitamin D insufficiency or deficiency, and what key symptoms should clinicians look for to differentiate between these conditions in their patients?
A: While vitamin D deficiency and insufficiency present with symptoms related to impaired bone health, such as muscle weakness, bone pain, and increased fracture risk, hypervitaminosis D manifests primarily through hypercalcemia-related symptoms. Patients with hypervitaminosis D may experience nausea, vomiting, constipation, abdominal pain, loss of appetite, polyuria, polydipsia, dehydration, weakness, and confusion. In severe cases, more serious complications like nephrolithiasis, nephrocalcinosis, acute kidney injury, and cardiac arrhythmias can occur. Differentiating these conditions requires thorough clinical evaluation, including dietary history, vitamin D supplementation usage, and laboratory assessments of 25(OH)D, calcium, phosphorus, and PTH. Consider implementing a standardized diagnostic approach for vitamin D-related disorders to enhance early detection and appropriate management. Learn more about the specific symptoms and long-term implications of untreated hypervitaminosis D.
Patient presents with symptoms suggestive of hypervitaminosis D, also known as vitamin D toxicity. Presenting complaints include nausea, vomiting, anorexia, abdominal pain, constipation, polyuria, polydipsia, and generalized weakness. The patient reports excessive intake of vitamin D supplements, specifically [Dosage and brand name if known] daily for [Duration]. Laboratory findings reveal elevated serum 25-hydroxyvitamin D levels exceeding 150 ngmL, confirming the diagnosis of vitamin D toxicity. Elevated serum calcium, hypercalcemia, and elevated serum phosphate levels are also noted. Differential diagnoses considered included primary hyperparathyroidism, sarcoidosis, and other causes of hypercalcemia. Treatment plan includes immediate discontinuation of all vitamin D supplements and dietary sources of vitamin D. Patient education provided regarding the importance of adhering to recommended vitamin D intake guidelines. Monitoring of serum calcium, phosphate, and 25-hydroxyvitamin D levels will be conducted to assess treatment response. Further investigation into potential complications of hypervitaminosis D, including nephrolithiasis, renal failure, and cardiac arrhythmias, will be undertaken if clinically indicated. Follow-up appointment scheduled in [Timeframe] to reassess clinical status and laboratory values. ICD-10 code E67.1, Overdosage of vitamin D, is assigned.