Find comprehensive information on hypogonadism diagnosis, including clinical documentation, medical coding (ICD-10-CM, SNOMED CT), symptoms, treatment, and lab tests. Learn about male hypogonadism, female hypogonadism, testosterone deficiency, estrogen deficiency, and related hormonal imbalances. This resource provides healthcare professionals with essential guidance on accurately documenting and coding hypogonadism for improved patient care and streamlined billing processes. Explore the latest research and best practices for managing hypogonadism in clinical settings.
Also known as
Testicular hypofunction
Male hypogonadism, encompassing primary and secondary causes.
Ovarian failure
Female hypogonadism related to ovarian dysfunction.
Delayed puberty, female
Hypogonadism can contribute to delayed puberty in females.
Delayed puberty, male
Hypogonadism may cause delayed pubertal development in males.
Follow this step-by-step guide to choose the correct ICD-10 code.
Is the hypogonadism due to a drug or other external agent?
Yes
Code E24.1, Drug-induced hypogonadism
No
Is it postprocedural?
When to use each related code
Description |
---|
Deficient sex hormone production |
Klinefelter syndrome (47,XXY) |
Turner syndrome (45,X) |
Coding E29.1 (Unspecified hypogonadism) without documenting the specific type (primary/secondary/drug-induced) leads to inaccurate data and potential claim denials. Use specific codes like E29.0, E29.8, E29.9 when applicable.
Miscoding age-related decline in testosterone as pathological hypogonadism (E29.-) can lead to inappropriate treatment and inflated prevalence statistics. Ensure proper documentation justifies diagnosis.
Failing to distinguish between late-onset (acquired) and congenital hypogonadism using appropriate codes like E29.0, Q98.0 impacts data analysis and quality reporting. Document the onset clearly.
Patient presents with concerns consistent with hypogonadism. Symptoms include [Specify symptoms e.g., decreased libido, erectile dysfunction, fatigue, muscle weakness, loss of body hair, decreased bone density, infertility, gynecomastia in males, hot flashes in males, irregular menses in females]. Onset of symptoms was [Specify timeframe e.g., gradual over several months, sudden onset following [event], etc.]. Patient reports [Specify relevant patient-reported history e.g., prior testicular injury, history of mumps orchitis, family history of Kallmann syndrome, exposure to toxins, chronic illness, use of medications such as opioids or glucocorticoids, chemotherapy or radiation exposure]. Physical examination reveals [Specify relevant findings e.g., small testicular volume in males, decreased muscle mass, increased body fat, sparse body hair]. Differential diagnosis includes primary hypogonadism, secondary hypogonadism, Klinefelter syndrome, Kallmann syndrome, pituitary adenoma, and medication-induced hypogonadism. Initial laboratory testing ordered includes [Specify tests e.g., serum testosterone levels free and total, LH, FSH, estradiol, prolactin, iron studies, CBC, CMP, lipid panel, bone density scan]. Treatment plan includes [Specify treatment plan e.g., testosterone replacement therapy TRT, referral to endocrinology, lifestyle modifications including diet and exercise, genetic counseling if indicated, addressing underlying medical conditions]. Patient education provided regarding the diagnosis, treatment options, potential benefits and risks of treatment, and the importance of follow-up care. Follow-up appointment scheduled in [Specify timeframe e.g., 4 weeks] to review laboratory results and assess treatment response. The patient verbalized understanding of the plan. ICD-10 code [Specify appropriate code e.g., E29.1, N95.1, Q98.2] is considered pending further diagnostic evaluation.