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I67.2
ICD-10-CM
Intracranial Atherosclerosis

Find information on intracranial atherosclerosis diagnosis, including clinical documentation requirements, ICD-10 codes (I67.2), medical coding guidelines, and healthcare resources. Learn about intracranial artery stenosis, cerebrovascular disease, stroke prevention, and treatment options. This resource offers support for accurate medical coding and billing related to intracranial atherosclerosis for healthcare professionals.

Also known as

Cerebral Atherosclerosis
Brain Artery Hardening

Diagnosis Snapshot

Key Facts
  • Definition : Narrowing of brain arteries due to plaque buildup, reducing blood flow.
  • Clinical Signs : Stroke, TIA (mini-stroke), dizziness, headache, cognitive impairment, numbness.
  • Common Settings : Neurology clinics, stroke centers, hospitals, primary care.

Related ICD-10 Code Ranges

Complete code families applicable to AAPC I67.2 Coding
I67.2

Cerebral atherosclerosis

Narrowing of brain arteries due to plaque buildup.

I65.2

Occlusion and stenosis of cerebral arteries

Blockage or narrowing of brain arteries, often from atherosclerosis.

I63.9

Cerebral infarction, unspecified

Stroke due to blockage of brain blood flow, sometimes caused by atherosclerosis.

I60-I69

Cerebrovascular Diseases

Diseases affecting blood vessels in the brain, including atherosclerosis.

Code-Specific Guidance

Decision Tree for

Follow this step-by-step guide to choose the correct ICD-10 code.

Is the intracranial atherosclerosis confirmed?

  • Yes

    Is there associated cerebral infarction?

  • No

    Do not code intracranial atherosclerosis. Code signs/symptoms or suspected diagnosis.

Code Comparison

Related Codes Comparison

When to use each related code

Description
Intracranial artery narrowing
Moyamoya disease
Vertebral artery stenosis

Documentation Best Practices

Documentation Checklist
  • Intracranial atherosclerosis ICD-10 code I67.2 documentation
  • Confirm stenosis location and laterality (e.g., right MCA)
  • Specify symptom onset: acute, subacute, chronic
  • Document diagnostic imaging results (CTA, MRA, DSA)
  • Include stroke risk factors: HTN, DM, smoking

Mitigation Tips

Best Practices
  • Document symptom onset, duration, and specific neurological deficits for accurate ICD-10 coding (I67.2).
  • Ensure consistent CDI of cerebrovascular events and risk factors (e.g., hypertension, diabetes) for complete HCC coding.
  • Use standardized terminology for intracranial stenosis severity (e.g., mild, moderate, severe) to improve data quality.
  • Timely follow-up imaging and documentation of treatment response aids appropriate CPT coding for compliance.
  • Detailed documentation of medical decision-making supports risk adjustment and quality reporting.

Clinical Decision Support

Checklist
  • 1. Verify ischemic stroke symptoms: sudden onset, focal neurologic deficits.
  • 2. Confirm intracranial stenosis 50-99% on imaging (CTA, MRA, DSA).
  • 3. Rule out other stroke etiologies: cardioembolism, large artery atherosclerosis.
  • 4. Document specific artery location and degree of stenosis for accurate ICD-10 coding (I67.2).
  • 5. Assess patient risk factors: hypertension, diabetes, smoking, dyslipidemia.

Reimbursement and Quality Metrics

Impact Summary
  • Intracranial Atherosclerosis: Reimbursement and Quality Metrics Impact Summary
  • Keywords: ICD-10 I67.2, Cerebral artery atherosclerosis, Stroke, TIA, Medical billing, Coding accuracy, Hospital quality reporting, Denials management, Risk adjustment
  • Impact 1: Accurate I67.2 coding maximizes appropriate reimbursement for stroke care.
  • Impact 2: Miscoding can lead to denials and lost revenue. Proper documentation is crucial.
  • Impact 3: Atherosclerosis impacts quality metrics related to stroke prevention and management.
  • Impact 4: Timely diagnosis and intervention improve patient outcomes and reduce readmissions.

Streamline Your Medical Coding

Let S10.AI help you select the most accurate ICD-10 codes for . Our AI-powered assistant ensures compliance and reduces coding errors.

Frequently Asked Questions

Common Questions and Answers

Q: What are the most effective diagnostic imaging modalities for confirming Intracranial Atherosclerosis in patients with suspected stroke or TIA?

A: While various imaging techniques can assess intracranial vessels, the gold standard for confirming Intracranial Atherosclerosis remains catheter angiography. However, due to its invasive nature, non-invasive techniques like Magnetic Resonance Angiography (MRA) and Computed Tomography Angiography (CTA) are often preferred as first-line options. MRA, particularly Time-of-Flight (TOF) MRA, offers good visualization of intracranial vessels and can detect stenosis and other vascular abnormalities. CTA provides excellent spatial resolution, allowing for precise assessment of arterial wall calcifications, a hallmark of Intracranial Atherosclerosis. Duplex ultrasound can be used for extracranial assessment, but its utility is limited for intracranial vessels. Explore how combining these modalities can offer a comprehensive evaluation and inform individualized treatment plans.

Q: How do I differentiate Intracranial Atherosclerosis from other causes of stroke or TIA, such as small vessel disease or cardioembolism, in my differential diagnosis?

A: Differentiating Intracranial Atherosclerosis from other stroke etiologies requires careful consideration of patient history, clinical presentation, and neuroimaging findings. While lacunar infarcts are characteristic of small vessel disease, Intracranial Atherosclerosis typically presents with territorial infarcts or TIA affecting larger vascular territories. Cardioembolism can also cause territorial infarcts, but often presents with multiple infarcts in different vascular territories or evidence of cardiac abnormalities. The location of stenosis on imaging is crucial; intracranial stenosis favors Intracranial Atherosclerosis, while extracranial stenosis may point to other etiologies. Consider implementing a structured diagnostic approach incorporating detailed imaging (MRA, CTA), cardiac evaluation (ECG, echocardiography), and risk factor assessment (hypertension, diabetes, smoking) to accurately diagnose Intracranial Atherosclerosis and guide tailored management strategies. Learn more about risk stratification tools for stroke prevention.

Quick Tips

Practical Coding Tips
  • Code I67.2 for intracranial atherosclerosis
  • Verify laterality: I67.20 - I67.23
  • Document specific artery affected
  • Confirm diagnosis with imaging studies
  • Consider sequelae, e.g., stroke

Documentation Templates

Patient presents with symptoms suggestive of intracranial atherosclerosis, including transient ischemic attack, stroke, or focal neurological deficits.  The patient reports [specific symptom, e.g., temporary loss of vision in the right eye, weakness in the left arm, difficulty speaking].  Risk factors for intracranial artery stenosis, such as hypertension, hyperlipidemia, diabetes mellitus, smoking history, and family history of stroke, were assessed.  Physical examination reveals [relevant neurological findings, e.g.,  normal cranial nerve examination, mild left-sided hemiparesis, decreased sensation in the left lower extremity].  Neuroimaging, specifically magnetic resonance angiography MRA or computed tomography angiography CTA of the head and neck, was ordered to evaluate for intracranial atherosclerotic disease.  Differential diagnosis includes other causes of stroke, such as cardioembolism, small vessel disease, and arterial dissection.  Preliminary imaging findings indicate [specific findings, e.g., stenosis of the right middle cerebral artery].  Based on the patient's clinical presentation, risk factors, and imaging findings, a diagnosis of intracranial atherosclerosis is suspected.  Treatment plan includes medical management with antiplatelet therapy such as aspirin or clopidogrel, statin therapy for lipid management, and aggressive control of vascular risk factors.  Further evaluation with digital subtraction angiography DSA may be considered for definitive diagnosis and potential endovascular intervention if indicated.  Patient education regarding lifestyle modifications, including diet, exercise, and smoking cessation, was provided.  Referral to neurology and vascular surgery for ongoing management and secondary stroke prevention.  Follow-up appointment scheduled to monitor symptoms, medication effectiveness, and disease progression.
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